protein kinase inhibitor fragment 14 22 (Tocris)
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protein kinase inhibitor fragment 14 22
Protein Kinase Inhibitor Fragment 14 22, supplied by Tocris, used in various techniques. Bioz Stars score: 93/100, based on 55 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/protein+kinase+inhibitor+fragment+14+22/PKA+Inhibitor+Fragment+(6-22)+amide/pm40147739-83-85-105
Average 93 stars, based on 55 article reviews
Protein Kinase Inhibitor Fragment 14 22, supplied by Tocris, used in various techniques. Bioz Stars score: 93/100, based on 55 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/protein+kinase+inhibitor+fragment+14+22/PKA+Inhibitor+Fragment+(6-22)+amide/pm40147739-83-85-105
Average 93 stars, based on 55 article reviews
protein kinase inhibitor fragment 14 22 - by Bioz Stars,
2026-09
93/100 stars
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Concentration Assay:Article Title: Early life seizures chronically disrupt L-type voltage gated calcium channel regulation of mGluR mediated long term depression via interactions with protein phosphatase 2A. Article Snippet: We probed the dependence of metabotropic glutamate receptor dependent long-term depression (mGluR-LTD) on L-type voltage gated calcium channels (LTCCs).. In prior work, we found that in a rat model of early life seizures (ELS), exaggerated mGluR-LTD was partly mediated by LTCCs and protein phosphatase 2A (PP2A).. Here, we further investigated the interactive role of LTCCs, PP2A, and protein kinase A (PKA) in this same model. PP2Ac is known to bind CaV1.2 and modulate its function; displacement of PP2A (C subunit, or PP2Ac) as well as PKA phosphorylation of CaV1.2 at serine 1928, result in enhanced CaV1.2 function. Blocking Assay:Article Title: Early life seizures chronically disrupt L-type voltage gated calcium channel regulation of mGluR mediated long term depression via interactions with protein phosphatase 2A. Article Snippet: We probed the dependence of metabotropic glutamate receptor dependent long-term depression (mGluR-LTD) on L-type voltage gated calcium channels (LTCCs).. In prior work, we found that in a rat model of early life seizures (ELS), exaggerated mGluR-LTD was partly mediated by LTCCs and protein phosphatase 2A (PP2A).. Here, we further investigated the interactive role of LTCCs, PP2A, and protein kinase A (PKA) in this same model. PP2Ac is known to bind CaV1.2 and modulate its function; displacement of PP2A (C subunit, or PP2Ac) as well as PKA phosphorylation of CaV1.2 at serine 1928, result in enhanced CaV1.2 function. |